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14th International Conference on Chronic Obstructive Pulmonary Disease , will be organized around the theme “”

COPD 2022 is comprised of keynote and speakers sessions on latest cutting edge research designed to offer comprehensive global discussions that address current issues in COPD 2022

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Chronic obstructive pulmonary disease (COPD) is a preventable and treatable disease in responsible for human and economic burden around the world. Cigarette smoking is the fundamental hazard factor for COPD in the developed world, although other essential respiratory disorder incorporate word related exposures, air contamination, airway route hyper responsiveness, asthma, and hereditary predisposition. In the vast majority of the world, Epidemiology of COPD prevalence and mortality keep on rising in light of increases in smoking addiction, especially by women and adolescents. COPD is likewise an imperative reason for disability, and is connected to comorbid diseases, for example, depression and cardiovascular disease, which adds to the large economic burden related with this issue. Better public health and medical intercessions that target both the risk factors for COPD and look toward prior mediation may decrease growing public health impact of COPD. COPD Conferences will focus on the understanding of all the pulmonary treatments.

  • Track 1-1Morbidity of COPD
  • Track 1-2Molecular and Genetic Risk Factors
  • Track 1-3Occupational Exposure
  • Track 1-4Mortality of COPD
  • Track 1-5Social and economic factors

The objective of COPD therapeutics is to enhance a patient's functional status and quality by protecting optimal lung function, enhancing indications, and keeping the recurrence of COPD exacerbations. At present, no medicines aside from lung transplantation have been appeared to significantly enhance lung function or decrease mortality; however, oxygen therapy and smoking cessation may decrease mortality. Once the diagnosis of COPD is established, it is essential to educate the patient about the sickness and to empower their active interest in treatment. Bronchodilators are the foundation of any COPD treatment regimen.They work by enlarging aviation routes, accordingly diminishing airsac protection. Pulmonary Conferences provides a multidisciplinary to pulmonology researchers.

  • Track 2-1Smoking Cessation
  • Track 2-2Corticosteroids
  • Track 2-3Oxygen Therapy
  • Track 2-4Pulmonary Rehabilitation

For Chronic Obstructive Pulmonary Disease there are no sign and symptoms or sometimes it may show mild symptoms. As the disease gets inferior, symptoms usually progress more severe. The first symptom of COPD is usually a long-term or chronic cough. If you have COPD, you also may often have colds or other respiratory infections such as the flu, or influenzaPulmonary Conferences discuss about the COPD symptoms, often do not appear until significant lung damage or lung cancer has occurred, and they usually worsen over time, particularly if smoking exposure continues. Symptoms include increasing breathlessness – this may just occur when exercising at first and you may sometimes wake up at night feeling breathless, a persistent chesty cough with phlegm that never seems to go away, frequent chest infections, persistent wheezing.

  • Track 3-1Shortness of breath
  • Track 3-2Wheezing
  • Track 3-3Chest tightness
  • Track 3-4Chronic Cough
  • Track 3-5Unintended weight loss

Even if an individual has never smoked or been unprotected to pollutants for an extended period, they can still develop COPDAlpha-1 Antitrypsin Deficiency (AATD) is the most commonly known genetic risk factor of COPD in emphysema. Alpha-1 Antitrypsin related COPD is caused by a deficiency of the Alpha-1 in the bloodstream. Without the Alpha-1 Antitrypsin protein, white blood cells begin to harm the lungs and lung deterioration occurs. The World Health Organization and the American Thoracic Society suggests that everyone determined to have COPD be tested for Alpha-1Pulmonary Conferences also discusses about different types of genes which may be a cause of pulmonary diseases.

  • Track 4-1Alpha1-antitrypsin
  • Track 4-2Alpha1-antichymotrypsin
  • Track 4-3Cystic fibrosis transmembrane regulator
  • Track 4-4Vitamin D-binding protein
  • Track 4-5Alpha2-macroglobulin

Additional pulmonary comorbidities impact the anticipation of patients with COPD. Tobacco smoking is a typical hazard factor for much comorbidity, including coronary illness, heart failure and lung cancer. Comorbidities, for example, pulmonary artery disease and malnutrition are specifically caused by COPD, while others, such as fundamental venous thromboembolism, anxiety, depression, osteoporosis, obesity, metabolic disorder, diabetes, sleep disturbance and anaemia, have no clear physiopathological association with COPD. Co-Morbidities of COPD shared conviction between the majorities of these extra pulmonary signs is chronic systemic inflammation. These diseases potentiate the morbidity of COPD, prompting expanded hospitalisations and healthcare costs. They can frequently cause death, independently of respiratory failure. Comorbidities make the management of COPD difficult and should be evaluated and treated adequately. COPD Conferences enlighten the recent advances in COPD and pulmonology research.

  • Track 5-1Cardiac Disease
  • Track 5-2Diabetes Mellitus
  • Track 5-3Hypertension
  • Track 5-4Osteoporosis
  • Track 5-5Psychological Disorders

Chronic obstructive pulmonary disease is one of the leading causes of disability and death worldwide. COPD exacerbation is usually treated with antibiotics, systemic corticosteroids, and inhaled bronchodilators. COPD exacerbation was treated repeatedly with standardtherapy. Dynamic expiratory computed tomography of the chest was done, which revealed concomitant tracheomalacia. COPD and tracheomalacia may coexist during recurrent exacerbations of COPD, and delayed diagnosis can be associated with severe comorbidities. Ordering the appropriate imaging technique may aid in the correct diagnosis and facilitate appropriate management. COPD Conferences will survey the evolution of pulmonology.

  • Track 6-1End-stage Lung Disease
  • Track 6-2Pulmonology
  • Track 6-3Bronchiectasis
  • Track 6-4Obstructive lung disease
  • Track 6-5Pulmonary Emphysema

Inflammation is present in the lungs, especially the small airway routes, of all people who smoke. COPD Pathogenesis is typical defensive reaction to the inhaled toxins is enhanced in COPD, leading to tissue destruction ,debilitation of the defence mechanisms and interruption of the repair mechanisms. In general, the inflammatory and structural changes in the airway routes increased with disease seriousness and hold on even after smoking cessation. Other than inflammation, two different procedures are associated with the pathogenesis of COPD an irregularity amongst  proteases  and  antiproteases  and an imbalance amongst  oxidants and antioxidants agents in the lungs. Pulmonary Conferences expresses various developing treatments for COPD.

  • Track 7-1Inflammatory mediators
  • Track 7-2Oxidative stress
  • Track 7-3Mucous Hypersecretion
  • Track 7-4Air trapping
  • Track 7-5Airway inflammation
  • Track 7-6Mucociliary dysfunction
  • Track 7-7Protease imbalance
  • Track 7-8Antiprotease imbalance

Bronchial Asthma  and Chronic Obstructive Pulmonary Disease are obstructive pneumonic sicknesses that influenced a large number of people everywhere throughout the world. Chronic Obstructive Pulmonary Disease (COPD) is a group of pulmonary diseases that block air stream in the lungs. Asthma is a chronic disease noticeable by spasms of bronchi, due to inflamed and narrowed airways in the lungs. Asthma causes difficulty in breathing that often results from an allergic reaction. There is large evidence that treatment with anti-inflammatory drugs reduces morbidity and mortality in asthma. Inhaled corticosteroids appear to have a place in the management of severe chronic obstructive pulmonary disease, perhaps by decreasing the frequency of exacerbations. Lung Conferences envelop the fields of asthma and COPD which have important similarities and differences. Asthma and COPD are chronic inflammatory diseases that include the little airway routes and cause airflow impediment, both result from gene environment communications and both are typically characterized by mucus and bronchoconstriction.

  • Track 8-1Airflow obstruction
  • Track 8-2Bronchospasm
  • Track 8-3Nocturnal Asthma
  • Track 8-4Chronic Obstructive Airways Disease

Constant Obstructive Pulmonary Disease (COPD) experiences long-term and dynamic damage to their lungs. This impacts air flow to the lungs. Doctors in some cases call this condition chronic bronchitis or chronic emphysema. Those with COPD can experience periods when their signs are much worse than normal. This is known as an acute exacerbationShortness of breath and chest tightness is present in many cases. A patient encountering a COPD exacerbation may need to search for medicinal help at a hospital. COPD exacerbations can be harmful because they can make damage to the lungs. COPD, keeping a compounding from happening can enable you to live a healthier life and diminish risk of death. Self-management and prevention for COPD will be beneficial for future exacerbation. Lung Conferences will explore the application of pulmonology.


  • Track 9-1Acute Exacerbation
  • Track 9-2Pathophysiology of exacerbations
  • Track 9-3Management of Exacerbation

People with different types of COPD experience difficulty in breathing and shortness of breath. The two principle infections that fall under the extent of COPD are: Chronic bronchitis, which involves a long-term cough with mucus, it is a long-term inflammation of the bronchi, which results in increased production of mucus, as well as other changes. These changes may result in breathing problems, frequent infections, cough, disability, anxiety and depression. Emphysema, which involves damage to the lungs over time. Emphysema is a chronic lung condition in which alveoli may be destroyed, narrowed, collapsed, stretched, and over-inflated. This can cause a reduction in respiratory capacity and breathlessness. Harm to the air sacs is irreversible and brings about permanent "holes" in the lung tissue. COPD Conferences will focus on the different techniques used in pulmonology.

  • Track 10-1Chronic Bronchitis
  • Track 10-2Emphysema
  • Track 10-3Pulmonary Emphysema
  • Track 10-4Obstructive lung disease

Patients with chronic obstructive pulmonary disease are at increased risk for both the development of primary lung cancer, as well as poor outcome after lung cancer diagnosis and treatment. Because of existing impairments in lung function, patients with COPD often do not meet traditional criteria for tolerance of definitive surgical lung cancer therapy. Emerging information with respect to the pathophysiology of COPD in lung resection demonstrates that postoperative decrements in lung capacity may be less than anticipated by traditional prediction tools. In patients with COPD, more inclusive consideration for surgical resection with curative intent might be appropriate as constrained surgical resections or nonsurgical therapeutic options provide inferior survival. Besides, optimizing perioperative COPD therapeutic care as indicated by clinical practice rules including smoking cessation can possibly minimize morbidity and enhance functional status in this often severely impaired patient population. Lung Conferences concentrates on the risk factor for development of lung cancer.

  • Track 11-1Small-Cell Lung Carcinoma
  • Track 11-2Prognosis of Lung Cancer
  • Track 11-3Genetic susceptibility
  • Track 11-4Chronic inflammation

Chronic obstructive pulmonary disease is related with increased risk of cardiovascular disease, for example, heart failure or a heart attack. The lungs and the heart work firmly together to supply the oxygen; oxygen in the air that comes into the lungs is moved into the circulation system, which the heart at that point pushes out to rest of the body. But diseases in both the heart and the lungs often go together. If person have COPD then there is a higher risk of having cardiovascular diseases. Comorbidities and chronic obstructive pulmonary disease (COPD) are pervasive, with cardiovascular disease being the most well-known and significant. Risk factors for COPD and Cardiovascular Diseases, such as smoking, low socioeconomic class, and a sedentary lifestyle contribute to the natural history of each of these conditions. COPD Conferences will focus on the scope of COPD related to cardiovascular diseases.

  • Track 12-1Pathogenesis
  • Track 12-2Vascular remodelling
  • Track 12-3Dynamic hyperinflation
  • Track 12-4Pulmonary artery catheterization
  • Track 12-5Respiratory Treatment and Dysrhythmias

Pulmonary hypertension is a type of high blood pressure that impacts the arteries in lungs and the right side of heart. In one type of pulmonary hypertension, modest arteries in lungs, called pulmonary arterioles, and capillaries become narrowed, blocked or destroyed. This makes it harder for blood to flow through lungs, and raises pressure inside the arteries of lungs. As the pressure builds, heart's lower right chamber (right ventricle) must work harder to direct blood through lungs, in the end causing heart muscle to debilitates and fail. Few categories of pulmonary hypertension are serious conditions that become progressively worse and sometimes become fatal. Pulmonary hypertension can lead to a number of COPD complications such as blood clots in the lungs, sickle cell disease and sleep apnea. Although a few kinds of pulmonary hypertension are not curable, treatment can help decrease symptoms and enhance quality of life. Pulmonary Conferences concentrates on the impact of pulmonary hypertension.

  • Track 13-1Pulmonary arterial hypertension
  • Track 13-2Pulmonary Embolism
  • Track 13-3Pulmonary Venous Hypertension
  • Track 13-4Molecular pathology
  • Track 13-5Acute Exacerbation

Chronic obstructive pulmonary disease (COPD) is a life-threatening condition. It affects lungs and ability to breathe. The pathophysiology of COPD is the physical changes associated with it, start with damage to airways and the air sacs in lungs. It progresses from a cough with mucus to difficulty breathing. To understand pathophysiology of COPD, it is essential to understand the structure of the lungs. When you inhale, air moves down in trachea through two tubes called bronchi. The bronchi branch out into smaller tubes called bronchioles. At the ends of the bronchioles are little air sacs called alveoli. And at the end of alveoli are capillaries, which are tiny blood vesselsLung Conferences envision the pulmonology.

  • Track 14-1Inflammatory mediators
  • Track 14-2Oxidative stress
  • Track 14-3Mucous Hypersecretion
  • Track 14-4Air trapping

Individuals with COPD can be in risk for serious complications that can not only put their health in jeopardy but can also be fatal. Cor Pulmonale of COPD bring down extremity edema (swelling) in a patient with COPD Complications is typically an indication of core pulmonale (pulmonary hypertension and right-sided heart failure). Acute COPD Exacerbations are portrayed by an unexpected increment of manifestations. Cough and sputum production increases. At the point when respiratory failure happens in a patient who slowly, there is a moderate decrease in lung capacity and rising levels of carbon dioxide in the blood. The expanding carbon dioxide makes an opiate impact in the patient, who gradually loses awareness and quits relaxing. Different difficulties of COPD incorporate pneumonia, polycythaemia, and pneumothorax. Pneumonia caused by bacterial disease can prompt respiratory failure in these patients. Streptococcus pneumoniae is the most well-known reason for bacterial pneumonia in patients with COPD. Pneumothorax happens when a hole develops in the lung, enabling air to escape into the space between the lung and the chest wall and collapsing the lung. Polycythaemia in COPD is the body's endeavour to adjust to decreased amount of blood oxygen by expanding the generation of oxygen-conveying red blood cells. While this might be useful temporarily, overproduction eventually clogs small blood vessels. COPD Conferences provides global forum for discussion about new innovation of pulmonology research.

  • Track 15-1End-stage Lung Disease
  • Track 15-2Cor Pulmonale
  • Track 15-3Pneumothorax
  • Track 15-4Lung cancer